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15th International Conference and Exhibition on Alzheimer’s Disease, Dementia & Aging, will be organized around the theme “Scientific Innovations, Research Ideologies & Clinical Practice in Alzheimer Disease & Dementia”

Euro Dementia Congress 2019 is comprised of keynote and speakers sessions on latest cutting edge research designed to offer comprehensive global discussions that address current issues in Euro Dementia Congress 2019

Submit your abstract to any of the mentioned tracks.

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Dementia is not a specific disease. It's an overall term that describes a wide range of symptoms associated with a decline in memory or other thinking skills severe enough to reduce a person's ability to perform everyday activities. Alzheimer's disease. Vascular dementia, which occurs after a stroke, is the second most common dementia type. But there are many other conditions that can cause symptoms of dementia, including some that are reversible, such as thyroid problems and vitamin deficiencies. Dementia is a syndrome that involves severe loss of cognitive abilities as a result of disease or injury. Dementia caused by traumatic brain injury is often static, whereas dementia caused by neurodegenerative disorders, such as Alzheimer's disease, is usually progressive and can eventually be fatal. Dementia can be grouped based on the part of brain being affected. it is roughly true that the earliest symptoms in "cortical" dementia include difficulty with high-level behaviour’s such as memory, language, problem-solving and reasoning; these functions tend to be less impaired in "subcortical" dementia.  Cortical dementia occurs because of damage in the cerebral cortex, the outer layer of the brain. They play a critical role in memory and language. The symptoms usually include severe memory loss. Alzheimer's and Creutzfeldt-Jakob disease are two forms of cortical dementia. Subcortical dementia occurs due to the damage to the part of the brain beneath the cortex. The person suffering from it can show changes in their speed of thinking. Usually, people with subcortical dementia don't have forgetfulness and language problems. Parkinson's disease, Huntington's disease, and HIV are two forms subcortical dementia.


  • Track 1-1Mixed dementia
  • Track 1-2Frontotemporal dementia
  • Track 1-3Neurogenesis in dementia
  • Track 1-4Global prevalence of dementia
  • Track 1-5Signs and symptoms of dementia
  • Track 1-6Signs and symptoms of dementia
  • Track 1-7Novel therapeutics technology
  • Track 1-8Novel therapeutics strategies for Dementia

Lewy body  is also known as dementia with Lewy bodies, with Lewy body dementia is the second most common type of progressive dementia after Alzheimer's disease dementia. Protein deposits, called Lewy bodies, develop in nerve cells in the brain regions involved in thinking, memory and movement (motor control).

Lewy body dementia is also causes a progressive decline in mental abilities. The people with Lewy body dementia may experience visual hallucinations and changes in the alertness and attention and other effects include Parkinson's disease-like symptoms such as rigid muscles, slow movement and tremors.


  • Track 2-1Risk factor of vascular dementia
  • Track 2-2Antipsychotic medications
  • Track 2-3Antipsychotic medications
  • Track 2-4Palliative care in vascular dementia
  • Track 2-5Autophagy
  • Track 2-6Synucleinopathies

Alzheimer's Disease is irreversible and decimates cerebrum cells, making thinking capacity and memory weaken. Alzheimer's infection is a dynamic, degenerative mind ailment and the most widely recognized type of dementia, a gathering of brain disorders that influence a man's memory, considering and capacity to connect socially. Dementia and Alzheimer's infection isn’t the same. Dementia is a general term used to depict indications that effect memory, execution of day by day exercises, and correspondence capacities. Alzheimer's infection is the most widely recognized kind of dementia.

Alzheimer's illness isn't a typical piece of maturing. We are leading exploration into the part of aggravation in Alzheimer's sickness to discover potential focuses for remedial mediation. The Dominantly Inherited Alzheimer Network is the main aim to discover biomarkers for Alzheimer's Diseases that are perceptible some time before the underlying clinical side effects. Members experience cerebrum checks and clinical evaluations and give blood and spinal liquid to explore. Presently utilizing hereditary linkage ways to deal with endeavor to distinguish these modifier qualities, which may give helpful focuses to the treatment of Alzheimer's sickness



  • Track 3-1Role of Plaques and Tangles
  • Track 3-2Diagnostic and Statistical Manual of Mental Disorders
  • Track 3-3Mental Diseases
  • Track 3-4Dominantly Inherited Alzheimer Network (DIAN) study
  • Track 3-5Causative Genetic Mutation

Parkinson's ailment (PD) is an unending and dynamic neurological disorders that influence engine work, causing bradykinesia, postural unsteadiness, inflexibility and tremor very still. Side effects grow progressively, making start finding troublesome. PD likewise has a few non-engine highlights, including psychological wellness issues, rest unsettling influences and autonomic aggravations. At introduce, there is no cure for PD. The treatment is, subsequently, gone from treating side effects. There are right now two fundamental ways to deal with enhanced dopaminergic action inside the mind: expanding dopamine emission with exogenous operators and focusing on related neurotransmission pathways. Levodopa is the regular metabolic antecedent of dopamine and is profoundly successful for treating PD indications. Side effects of Parkinson's Diseases are normally overseen utilizing a specific dopamine receptor agonist. While these medications are valuable in beginning time Parkinson's, they tend to lose viability in later sickness stages


  • Track 4-1Prevention of Parkinson’s Disease by Immune gene
  • Track 4-2Disease beyond Genetic Mutations

Vascular dementia is the second most regular reason for dementia after Alzheimer's Diseases. Vascular changes that begin in mind territories that assume a key part in putting away and recovering data may cause memory misfortune that looks especially like Alzheimer's illness. Vascular dementia is a decrease in intuition abilities caused by conditions that square or diminish blood stream to the cerebrum, denying mind cells of fundamental oxygen and supplements. Deficient blood stream can harm and, in the end, dead cells anyplace in the body. The mind has one of the body's richest systems of veins and is particularly helpless. In vascular dementia, changes in speculation abilities now and then happen abruptly following strokes that square significant cerebrum veins. A developing number of specialists lean toward the expression "Vascular Cognitive Impairment (VCI)" to "vascular dementia”. A few examinations have discovered that vascular changes and other cerebrum irregularities may associate in ways that improve the probability of dementia finding. The analysis of dementia or gentle psychological impedance is affirmed by neurocognitive testing, which includes a few hours of composed or mechanized tests that give point by point assessment of reasoning abilities, for example, judgment, arranging, critical thinking, thinking and memory


  • Track 5-1Frontotemporal Dementia
  • Track 5-2Subcortical vascular dementia
  • Track 5-3Vascular Contributions to Cognitive Impairment and Dementia
  • Track 5-4Post-stroke dementia
  • Track 5-5Multi-infarct dementia
  • Track 5-6Pathobiology of vascular dementia

Brain diseases come in different forms of Infections, trauma, stroke, seizures, and tumors are some of the major categories of brain diseases. Here's an overview of various diseases of the brain

  • Track 6-1Migraine
  • Track 6-2Attention deficit hyperactivity disorder
  • Track 6-3Motor neuron disease
  • Track 6-4Bipolar disorder
  • Track 6-5Arteriovenous malformation
  • Track 6-6Encephalitis
  • Track 6-7Seizures
  • Track 6-8Trauma
  • Track 6-9Brain abscess

New research recommends there is a solid relationship between initial childhood trauma, for example, that accomplished by the stolen ages, and dementia. There is an expansive agreement demonstrating that childhood trauma is fundamentally associated with the improvement of sorrow. There is a solid connection between a high introduction to early life stress and affliction, that incorporates individuals that were a piece of the stolen ages, and the probability of being determined to have dementia sometime down the road. Psychic trauma is any psychological disorders that unexpectedly overpowers the ability to give a negligible feeling of wellbeing and integrative wholeness, bringing about overpowering nervousness or defenselessness, or its danger, and creating a continuing change in the psychic organization. Childhood trauma can influence mental and physical wellbeing into adulthood


  • Track 7-1Childhood Trauma and PTSD Symptoms
  • Track 7-2Psychological Therapies
  • Track 7-3Childhood trauma with cognitive function in healthy adults
  • Track 7-4Post-traumatic stress disorder and Alzheimer’s disease

Neurological disorders are maladies of the focal and fringe sensory system. At the end of the day, spinal rope, cranial nerves, fringe nerves, nerve roots, autonomic sensory system and neuromuscular intersection. These disarranges incorporate epilepsy, Alzheimer illness and different dementias, cerebrovascular sicknesses including stroke, headache and other migraine issue, various sclerosis, Parkinson's infection, neuroinfectious, mind tumors, awful scatters of the sensory system because of head injury, and neurological issue due to hunger. Neurological manifestations may happen because of the contamination itself or because of a safe reaction. Countless individuals worldwide are influenced by neurological disarranges. Alzheimer's sickness is the most widely recognized reason for dementia and may add to 60– 70% of cases

  • Track 8-1Chronic Neurological Diseases
  • Track 8-2Neurocutaneous Syndromes
  • Track 8-3Epilepsy and Seizures
  • Track 8-4Cerebral Aneurysm
  • Track 8-5Muscular Dystrophy

Neurodegenerative sicknesses are crippling, and it is a heterogeneous gathering of disarranges of serious conditions that outcome in dynamic degeneration. Neurodegenerative maladies cause issues with development called ataxias, or mental working called dementias. Neurodegenerative infections happen when nerve cells in the mind or fringe sensory system lose work after some time and at last pass on. Although medicines may help diminish a portion of the physical or mental side effects related with neurodegenerative infections, there is right now no cure or approach to moderate ailment movement. The danger of being influenced by a neurodegenerative increment drastically with age. A greatly little extent under five level of neurodegenerative infections are caused by hereditary changes. This makes a basic need to enhance our comprehension of what causes neurodegenerative infections and grow new methodologies for treatment and counteractive action

  • Track 9-1Alzheimer’s disease
  • Track 9-2Huntington’s disease
  • Track 9-3Amyotrophic lateral sclerosis (ALS)
  • Track 9-4Motor neuron disease

Vascular insults can start a course of Molecular event occasions prompting neurodegeneration, psychological disorders, and dementia. Here, the molecular mechanisms and cell in cerebral veins pathophysiological occasions prompting disturbance of the blood-cerebrum obstruction and cerebral blood stream dysregulation and the neurovascular unit, which all may add to the beginning and movement of dementia and Alzheimer's disease. From the most punctual phases of improvement, when cerebral neurogenesis and angiogenesis are weaved, to the finish of life, the interaction between neural frameworks and vascular of the cerebrum is basic in wellbeing and infection. Cerebral blood stream control is fundamental for ordinary mind work. The mammalian mind has developed an exceptional system for CBF control known as neurovascular coupling.


  • Track 10-1Neurovascular pathways to neurode-generation
  • Track 10-2Neurovascular regulation in the normal brain
  • Track 10-3Cerebral microvasculature
  • Track 10-4Dysfunction of the blood–brain barrier
  • Track 10-5Neurovascular coupling
  • Track 10-6Dermal Neurovascular Dysfunction

Alzheimer’s disease Biomarkers, with emphasis on cerebrospinal fluid biomarkers and neuroimaging. Mild Cognitive Impairment (MCI) of Neuroimaging-biomarkers allow an early diagnosis in preclinical stages of Alzheimer’s disease. Biomarker changes in patients with late-onset AD, including findings from studies using structural and functional magnetic resonance imaging (MRI), advanced MRI techniques (diffusion tensor imaging, magnetic resonance spectroscopy, perfusion), positron emission tomography with fluorodeoxyglucose, amyloid tracers, and other neurochemical tracers, and CSF protein levels. The dynamic measures of these imaging biomarkers are used to predict the disease progression in the early stages and improve the assessment of therapeutic efficacy in these diseases in future clinical trials


  • Track 11-1Neurogenesis & Neuron Regeneration
  • Track 11-2Structural Neuroimaging
  • Track 11-3Neuropathology
  • Track 11-4Cerebrospinal fluid biomarker
  • Track 11-5Structural and functional biomarkers

Neuropharmacology is an extremely wide locale of science that includes numerous parts of the sensory system from single neuron control to whole ranges of the cerebrum, spinal line, and fringe nerves. To better comprehend the premise behind medication advancement, one should first see how neurons speak with each other. This article will concentrate on both behavioral and atomic neuropharmacology; the real receptors, particle channels, and neurotransmitters controlled through medication activity and how individuals with a neurological issue advantage from this medication activity.


  • Track 12-1Neuroscience and neuropharmacology
  • Track 12-2Neurochemical interaction
  • Track 12-3Molecular neuropharmacology
  • Track 12-4Behavioral neuropharmacology
  • Track 12-5Advance research in neuropharmacology

People with vascular dementia have different mental element shortfalls that incorporate every memory hindrance, that influences the adaptability to discover new data or review data already learned, and one or extra of the ensuing side effects aphasia, apraxia, agnosia, or official brokenness to such an extent that the mental element shortages adversely affect social or action working with a major decrease in past abilities. Furthermore, people with dementia commonly experience the ill effects of comorbid conditions that extra confuse mind and block best results. Along these lines, creating caregiving techniques individuals with vascular dementia is pressing, given this expanding commonness and consequently the related weight that dementia places not just on the people, however on the parental figures, relations, and thusly the assets of the human services framework. Traditional perspectives bearing on geriatric nursing ordinarily paint a picture of the care as being moderate paced certain and less requesting than intense care. Be that as it may, care of the matured, and especially those with vascular dementia, is normally confounded, unusual, and flimsy

  • Track 13-1Dementia nursing care plan
  • Track 13-2Music therapy in dementia
  • Track 13-3Physiotherapy for dementia
  • Track 13-4Clinical features of dementia
  • Track 13-5Therapeutic interventions in dementia

Most continuous endeavors to battle Alzheimer Disease are centered around treating its clinical symptoms, however the neuropathologic changes fundamental  show up decades sooner and turn out to be basically irreversible when the sickness achieves its clinical stages.This requires treating at preclinical stages, which requires a superior comprehension of the essential instruments prompting Alzheimer Disease pathology. Alzheimer's infection is a complex hereditary issue that is the main source of dementia in the elderly. Atomic confirmation raised the presumptions that trafficking of the amyloid precursor protein is under control of insulin flagging. Most progressing endeavors to battle Alzheimer infection are centered around treating its clinical side effects, however the neuropathologic changes fundamental show up decades sooner and turn out to be basically irreversible when the ailment achieves its clinical stages.


  • Track 14-1Fixing Genetic risk for Alzheimer’s
  • Track 14-2Medial Temporal Lobe
  • Track 14-3Nervous System Disease
  • Track 14-4Neurofibrillary Degeneration

The brain ordinarily therapists to some degree in solid maturing in any case, shockingly, does not lose neurons in huge numbers. In Alzheimer's sickness, be that as it may, harm is across the board, the same number of neurons quit working, lose associations with different neurons, and pass on. Alzheimer's disturbs forms fundamental to neurons and their systems, including correspondence, digestion, and repair. Acetylcholine is a neurotransmitter that is insufficient in individuals with Alzheimer's, the place beginning memory misfortune is trailed by more far reaching changes in considering and conduct that prompt lost freedom. Numerous molecular and cell changes happen in the mind of a man with Alzheimer's illness


  • Track 15-1Amyloid Plaques
  • Track 15-2Neurofibrillary Tangles
  • Track 15-3Chronic Inflammation
  • Track 15-4Vascular Contributions to Alzheimer’s Disease
  • Track 15-5Loss of Neuronal Connections and Cell Death

To evaluate the safety and efficacy of an oral investigational drug in individuals who are asymptomatic and at-risk for Alzheimer's disease and investigating the effect of crenezumab infusion on Alzheimer's disease progression in individuals with prodromal or mild Alzheimer's disease. Presently the clinical trials and studies are mainly concentrates on Alzheimer’s Disease & Down syndrome, aging to have the best chance of changing the outlook for people with dementia promising ideas must be driven through the development process and into clinical trials in people as quickly as possible

  • Track 16-1Targeting Amyloid
  • Track 16-2Ramping Up the Immune System
  • Track 16-3Non-Medication Approaches
  • Track 16-4Treatment trials
  • Track 16-5Diagnostic studies

Alzheimer's Disease as a dynamic and fatal neurodegenerative disease speaks to an immense neglected requirement for treatment. The low viability of current treatment techniques isn't just because of low medication intensity yet in addition because of the nearness of different hindrances in the conveyance courses. One of the fundamental boundaries is the blood brain boundary. The expanding predominance of Alzheimer's Diseases and the low viability of ebb and flow treatments have expanded the measure of research on unwinding of sickness pathways and advancement of treatment techniques. One of the fascinating regions for the last subject is biotechnology and their applications. Nanotechnology is a standout amongst the most vital field in biotechnology and has a noteworthy part in the improvement of novel remedial modalities with expanded adequacy. In the nanotechnology field, materials are designed with utilitarian association on the nanoscale. Every one of the materials are utilized for association and incitement of organic frameworks at the atomic level to instigate physiological reactions. One of the primary issues concerning the treatment of neurodegenerative diseases is identified with medicate conveyance


  • Track 17-1Alzheimer disease drug discovery
  • Track 17-2Molecular and Cellular Basis of Neurodegeneration
  • Track 17-3Vaccine against Alzheimer’s disease
  • Track 17-4Biology of Ageing

Doctors can't analyze Alzheimer's Disease until after death, when they can nearly inspect the brain under a magnifying instrument. However, they can utilize tests to decide out different conditions that may cause similar symptoms. Early, exact finding is advantageous for a few reasons. Starting treatment ahead of schedule in the illness procedure may help protect day by day working for quite a while, even though the hidden Alzheimer's procedure can't be ceased or switched. Alzheimer's dementia comes about because of the dynamic misfortune (degeneration) of brain cells. This movement may appear in an assortment of courses in brain scans. At present researchers are exploring a few ailment markers and demonstrative tests, for example, qualities, infection related proteins and imaging methods, which may precisely and dependably show whether you have Alzheimer's dementia or not. whatever, right now more research on these tests is important for future


  • Track 18-1Diagnostic Tests
  • Track 18-2Neuropsychological testing
  • Track 18-3Treatments for Behavior
  • Track 18-4Biomarker Research
  • Track 18-5Precision Medicine for Alzheimer Disease

Molecular biological research has considerably deepened our understanding of the pathophysiological basis of Alzheimer's dementia. Considerable progress has been made in our understanding of the genetics and molecular biology of dementia. Some genetic risk factors have been identified so far, but only a small proportion of AD cases can be explained by specific gene mutations. Genes may be related to disease in two ways: through autosomal-dominant mutations, in themselves sufficient to cause the disease alternatively, gene variations (polymorphisms) may indirectly increase disease risk without being sufficient in themselves to cause the disorder

  • Track 19-1Molecular Genetics of Frontotemporal Dementia
  • Track 19-2Molecular Genetics of Neurodegenerative Dementias
  • Track 19-3Human stem cell models of dementia
  • Track 19-4Fixing Genetic risk for Alzheimer’s

Caring for a person with dementia influences distinctive individuals in various ways. In any case, caring can also be both physically and rationally debilitating. It influences all aspects of your life and can influence you to feel separated, pushed and at times even discouraged. You may also have your own physical and psychological wellness needs, which you and others may neglect when you are tending to another person. There are numerous positive things about caring. These incorporate adapting new aptitudes, expanding on existing ones, reinforcing connections, having a feeling of pride in what you are doing, and supporting somebody who is imperative to you. Furthermore, because a man has dementia, it doesn't mean there won't in any case be great circumstances for you to share


  • Track 20-1Dealing with Dementia Behavior
  • Track 20-2Dementia Risk and Preventions

Novel therapeutics is a standout amongst the most energizing uses of data preparing frameworks is in diagnostics and treatment. This is not astonishing, given that illness analysis is in its center a data handling undertaking that finishes with a choice. In numerous infections, the conclusion can be performed in singular cells

  • Track 21-1Novel therapeutics strategies for Dementia
  • Track 21-2Novel therapeutics technology
  • Track 21-3Novel therapeutics molecule
  • Track 21-4Anti-dementia drugs
  • Track 21-5Novel drug target for the treatment of dementia

It is a branch of Medical Sciences majorly deals with Neuro tumors. Deals with Studies related to Brain and Spinal cord neoplasms. Neurooncology and Pediatric Neuro-oncology are the two different Concepts that differentiate the determining methodology of Neuro tumors. Neuro-oncology mainly includes especially related topics like Radiation therapy, Neurosurgery, Neuroimaging, social, Psychological, Neuropathology and psychiatric aspects.


  • Track 22-1Spine Cancer and Spine Cancer Treatment
  • Track 22-2Risk Factors
  • Track 22-3Neurotoxicity
  • Track 22-4Astrocytoma
  • Track 22-5Pediatric Neuro Oncology
  • Track 22-6Malignant Brain Cancers and Brain Metastasis
  • Track 22-7Brain Stem Tumors
  • Track 22-8Meningioma
  • Track 22-9Glioblastoma
  • Track 22-10Radiation Therapy And Chemotherapy